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001435601 001__ 1435601
001435601 003__ OCoLC
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001435601 020__ $$a9783030630461$$q(electronic bk.)
001435601 020__ $$a3030630463$$q(electronic bk.)
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001435601 0247_ $$a10.1007/978-3-030-63046-1$$2doi
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001435601 049__ $$aISEA
001435601 050_4 $$aRC771
001435601 08204 $$a616.2/41$$223
001435601 24500 $$aLung inflammation in health and disease.$$nVolume I /$$cYong-Xiao Wang, editor.
001435601 260__ $$aCham :$$bSpringer,$$c2021.
001435601 300__ $$a1 online resource (412 pages)
001435601 336__ $$atext$$btxt$$2rdacontent
001435601 337__ $$acomputer$$bc$$2rdamedia
001435601 338__ $$aonline resource$$bcr$$2rdacarrier
001435601 347__ $$atext file
001435601 347__ $$bPDF
001435601 4901_ $$aAdvances in Experimental Medicine and Biology ;$$vv. 1303
001435601 5050_ $$a1. Potential role of mast cells in regulating corticosteroid insensitivity in severe asthma -- 2. Galectin-3 Promotes ROS, Inflammation and Vascular Fibrosis in Pulmonary Arterial Hypertension -- 3. Anti-Inflammatory Effects of Statins in Lung Vascular Pathology: From Basic Science to Clinical Trials -- 4. Evolving Schema for Employing Network Biology Approaches to Understand Pulmonary Hypertension -- 5. Pulmonary Inflammation and KRAS Mutation in Lung Cancer -- 6. microRNA Targets for Asthma Therapy -- 7. Roles of genetic predisposition in the sex-bias of pulmonary pathophysiology, as a function of estrogens -- 8. Hypercapnic respiratory failure-driven skeletal muscle dysfunction: It is time for animal mod-els-based mechanistic research -- 9. Role of airway smooth muscle in inflammation related to asthma and COPD -- 10. Systemic Sclerosis and Pulmonary Disease -- 11. Innate Lymphoid Cells in airway inflammation -- 12. Sjogren's Syndrome and pulmonary disease -- 13. Redox Regulation, Oxidative Stress and Inflammation in Group 3 Pulmonary Hypertension -- 14. Sex-Steroid Signaling in Lung Diseases and Inflammation -- 15. Cytokines, Chemokines and Inflammation in Pulmonary Arterial Hypertension -- 16. Interactive roles of CaMKII/Ryanodine Receptor Signaling and Inflammation in Lung Diseases -- 17. Reciprocal Correlations of Inflammatory and Calcium Signaling in Asthma Pathogenesis -- 18. Crosstalk between Lung and Extrapulmonary Organs in Infection and Inflammation -- 19. Inflammation in Pulmonary Arterial Hypertension -- 20. Lysophospholids in Lung Inflammatory Diseases.
001435601 506__ $$aAccess limited to authorized users.
001435601 520__ $$aRespiratory diseases are leading causes of death and disability globally, with about 65 million people suffering from COPD, and 334 million from asthma, the most common chronic disease. Each year, tens of millions of people develop and can die from respiratory infections such as pneumonia and TB. Systemic inflammation may induce and exacerbate local inflammatory diseases in the lungs, and local inflammation can in turn cause systemic inflammation. There is increasing evidence of the coexistence of systemic and local inflammation in patients suffering from asthma, COPD, and other lung diseases, and the co-morbidity of two or more local inflammatory diseases often occurs. For example, rheumatoid arthritis frequently occurs together with, and promotes the development of, pulmonary hypertension. This co-morbidity significantly impacts quality of life, and can result in death for those affected. Current treatment options for lung diseases are neither always effective, nor often condition-specific; there is a desperate need for novel therapeutics in the field. Additionally, the molecular and physiological significance of most major lung diseases are not well understood, which further impedes development of new treatments, especially in the case of coexistent lung diseases with other inflammatory diseases. Great progress has been made in recent years in many areas of the field, particularly in understanding the molecular geneses, regulatory mechanisms, signalling pathways, and cellular processes within lung diseases, as well as basic and clinical technology, drug discovery, diagnoses, treatment options, and predictive prognoses. This is the first text to aggregate these developments. In two comprehensive volumes, experts from all over the world present state-of-the-art advances in the study of lung inflammation in health and disease. Contributing authors cover well-known as well as emerging topics in basic, translational, and clinical research, with the aim of providing researchers, clinicians, professionals, fellows, and students with new perspectives and concepts. The editor hopes this book will also help to direct future research in lung disease and other inflammatory diseases, and result in the development of novel therapeutics.
001435601 588__ $$aOnline resource; title from PDF title page (SpringerLink, viewed April 13, 2021).
001435601 650_0 $$aPneumonia.
001435601 650_0 $$aLungs$$xDiseases.
001435601 650_6 $$aPneumonie.
001435601 650_6 $$aPoumons$$xMaladies.
001435601 655_0 $$aElectronic books.
001435601 7001_ $$aWang, Yong-Xiao,$$eeditor.
001435601 77608 $$iPrint version:$$aWang, Yong-Xiao.$$tLung Inflammation in Health and Disease, Volume I.$$dCham : Springer International Publishing AG, ©2021$$z9783030630454
001435601 830_0 $$aAdvances in experimental medicine and biology ;$$vv. 1303.
001435601 852__ $$bebk
001435601 85640 $$3Springer Nature$$uhttps://univsouthin.idm.oclc.org/login?url=https://link.springer.com/10.1007/978-3-030-63046-1$$zOnline Access$$91397441.1
001435601 909CO $$ooai:library.usi.edu:1435601$$pGLOBAL_SET
001435601 980__ $$aBIB
001435601 980__ $$aEBOOK
001435601 982__ $$aEbook
001435601 983__ $$aOnline
001435601 994__ $$a92$$bISE