Molecular mechanisms of angiogenesis [electronic resource] : from ontogenesis to oncogenesis / Jean-Jacques Feige, Gilles Pagès, Fabrice Soncin, editors.
2014
QP106.6
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Title
Molecular mechanisms of angiogenesis [electronic resource] : from ontogenesis to oncogenesis / Jean-Jacques Feige, Gilles Pagès, Fabrice Soncin, editors.
ISBN
9782817804668 electronic book
281780466X electronic book
9782817804651
281780466X electronic book
9782817804651
Published
Paris : Springer, 2014.
Language
English
Description
1 online resource (xi, 505 pages) : illustrations (some color)
Item Number
10.1007/978-2-8178-0466-8 doi
Call Number
QP106.6
Dewey Decimal Classification
612.1/3
Summary
Angiogenesis is a multi-stage process that drives the generation of new blood and lymphatic vessels from pre-existing ones. It is highly active during embryogenesis, largely inactive during adulthood but reactivated during wound healing and under a number of pathological conditions including cancer and ocular diseases. In addition to endothelial cells, which line the walls of the vessels, several other cell types (pericytes, macrophages, progenitor cells) also contribute to angiogenesis. A number of signaling pathways are activated and very finely tune the delicate morphogenetic events that ultimately lead to the formation of stable bloodproof neovessels. This book reviews recent advances in our understanding of the molecular and cellular mechanisms of angiogenesis, with a focus on how to integrate these observations into the context of developmental, post-natal and pathological neovascularization. The book was published under the auspices of the French Angiogenesis Society. Most contributors are prominent members of this Society or international researchers who have actively contributed to the Annual Meetings of the Society.
Bibliography, etc. Note
Includes bibliographical references and index.
Access Note
Access limited to authorized users.
Source of Description
Description based on online resource; title from PDF title page (SpringerLink, viewed June 4, 2014).
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