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1 Models for Studying Organogenetic Gene Networks in the 21st Century; Abstract; 1.1 A Brief Historical Frame; 1.2 Choosing an Organogenetic Gene Network. Where to Start?; References; 2 Organogenesis of the C. elegans Vulva and Control of Cell Fusion; Abstract; 2.1 Background; 2.1.1 The Vulva of C. elegans as a Genetic Model Organ; 2.1.1.1 Historic Overview of Vulva Research; 2.1.2 Overview of Vulva Development; 2.2 Three Signaling Pathways Involved in the Control of Vulval Development; 2.2.1 Wnt Signaling; 2.2.2 Notch Signaling; 2.2.3 RTK-Ras-ERK
2.3 Formation and Maintenance of the Vulval Competence Group2.3.1 How Are the P Cells Polarized to Form Epidermal and Neuronal Linages?; 2.3.2 Notch Signaling and the Formation of the Anchor Cell; 2.3.3 Wnt and RTK/Ras/MAPK Signaling Maintain the Competence of the VPCs; 2.3.4 The Molecular Mechanism Involved in the Maintenance of Cell Cycle Quiescence During Late L1 and L2; 2.4 Vulval Cell Proliferation and Differentiation; 2.4.1 Current Understanding of VPC Fate Determination; 2.4.2 VPC Polarization and Longitudinal Divisions
2.4.3 The Third Division of the VPCs and Differentiation of Adult Vulval Cells2.4.4 VPC Fate Determination Research and Modeling; 2.5 Vulval Morphogenesis; 2.5.1 Formation of the Uterine-Vulval Connection; 2.5.2 Migration of the Vulval Cells Towards the Center of the Developing Vulva, Invagination and Formation of Seven Stacked Vulval Toroids; 2.5.3 Migration, Attachment and Innervation of Muscle Cells; 2.6 Insight into the Evolution of Vulva Development; 2.6.1 Variation in the Size of the Vulval Competence Group; 2.6.2 Reproductive Barriers; 2.6.3 Induction by Wnt Signaling; Acknowledgments
3.2.7 Programmed Cell Death During Drosophila CNS Development3.3 Drosophila Neuropeptide Neurons; Repertoire and Generation; 3.3.1 Neuropeptide Neurons: Common Properties and Specification Mechanisms Mediated by Dimmed; 3.3.2 Neuropeptide Neurons: Distinct Sub-types; 3.3.3 Specifying Neuropeptide Neurons; FMRFamide and Nplp1; 3.3.4 Specifying Neuropeptide Neurons; Leucokinin; 3.3.5 Specifying Neuropeptide Neurons; Corazonin; 3.3.6 Specifying Neuropeptide Neurons; Crustacean Cardioactive Peptide; 3.3.7 Specifying Neuropeptide Neurons; Capability
2.3 Formation and Maintenance of the Vulval Competence Group2.3.1 How Are the P Cells Polarized to Form Epidermal and Neuronal Linages?; 2.3.2 Notch Signaling and the Formation of the Anchor Cell; 2.3.3 Wnt and RTK/Ras/MAPK Signaling Maintain the Competence of the VPCs; 2.3.4 The Molecular Mechanism Involved in the Maintenance of Cell Cycle Quiescence During Late L1 and L2; 2.4 Vulval Cell Proliferation and Differentiation; 2.4.1 Current Understanding of VPC Fate Determination; 2.4.2 VPC Polarization and Longitudinal Divisions
2.4.3 The Third Division of the VPCs and Differentiation of Adult Vulval Cells2.4.4 VPC Fate Determination Research and Modeling; 2.5 Vulval Morphogenesis; 2.5.1 Formation of the Uterine-Vulval Connection; 2.5.2 Migration of the Vulval Cells Towards the Center of the Developing Vulva, Invagination and Formation of Seven Stacked Vulval Toroids; 2.5.3 Migration, Attachment and Innervation of Muscle Cells; 2.6 Insight into the Evolution of Vulva Development; 2.6.1 Variation in the Size of the Vulval Competence Group; 2.6.2 Reproductive Barriers; 2.6.3 Induction by Wnt Signaling; Acknowledgments
3.2.7 Programmed Cell Death During Drosophila CNS Development3.3 Drosophila Neuropeptide Neurons; Repertoire and Generation; 3.3.1 Neuropeptide Neurons: Common Properties and Specification Mechanisms Mediated by Dimmed; 3.3.2 Neuropeptide Neurons: Distinct Sub-types; 3.3.3 Specifying Neuropeptide Neurons; FMRFamide and Nplp1; 3.3.4 Specifying Neuropeptide Neurons; Leucokinin; 3.3.5 Specifying Neuropeptide Neurons; Corazonin; 3.3.6 Specifying Neuropeptide Neurons; Crustacean Cardioactive Peptide; 3.3.7 Specifying Neuropeptide Neurons; Capability