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Table of Contents
1. Advances in oral mucosal immunity and the microbiome
2. Trained innate immunity and its implications for mucosal immunity and inflammation
3. Signaling systems in oral bacteria
4. Origin of Th17 cells in Type 2 Diabetes-Potentiated Periodontal Disease
5. Gingival epithelial cell recognition of lipopolysaccharide
6. The relationship of Candida albicans with the oral bacterial microbiome in health and disease
7. A potential role of Phospholipase 2 group IIA (PLA2-IIA) in P. gingivalis-induced oral dysbiosis
8. Helicobacter spp. in experimental models of colitis
9. T Helper 17 cells as pathogenic drivers of periodontitis
10. Candida-bacterial biofilms and host-microbe interactions in oral diseases
11. Comparative analysis of gene expression patterns for oral epithelium-related functions with aging
12. Neutrophil interaction with emerging oral pathogens: a novel view of the disease paradigm
13. Biologically-defined or biologically-informed traits are more heritable than clinically-defined ones: the case of oral and dental phenotypes.
2. Trained innate immunity and its implications for mucosal immunity and inflammation
3. Signaling systems in oral bacteria
4. Origin of Th17 cells in Type 2 Diabetes-Potentiated Periodontal Disease
5. Gingival epithelial cell recognition of lipopolysaccharide
6. The relationship of Candida albicans with the oral bacterial microbiome in health and disease
7. A potential role of Phospholipase 2 group IIA (PLA2-IIA) in P. gingivalis-induced oral dysbiosis
8. Helicobacter spp. in experimental models of colitis
9. T Helper 17 cells as pathogenic drivers of periodontitis
10. Candida-bacterial biofilms and host-microbe interactions in oral diseases
11. Comparative analysis of gene expression patterns for oral epithelium-related functions with aging
12. Neutrophil interaction with emerging oral pathogens: a novel view of the disease paradigm
13. Biologically-defined or biologically-informed traits are more heritable than clinically-defined ones: the case of oral and dental phenotypes.